Patients › Hip

Greater trochanteric pain syndrome

Updated Sep 2026

What you're feeling¶

Greater trochanteric pain syndrome is a group of problems that cause pain on the outside of your hip. The name covers a few different causes: an irritated or inflamed bursa (a small fluid-filled cushion over the bony point of your hip), tears in the gluteal tendons (the strong cords that attach your buttock muscles to that bone), and snapping around the hip. More than one of these can be present at the same time.

The pain sits over the bony point on the side of your hip. Pressing on that spot is usually tender. Your hip may also feel weak when you move your leg out to the side, which is the movement your buttock muscles control. The pain often flares at night, especially when you lie on that side. It can also flare after activity, or first thing in the morning when you get out of bed.

Daily tasks that load the outside of your hip become hard. Walking, climbing stairs, standing on one leg to dress, and getting up from a chair can all hurt. Lying on the sore side at night may wake you or stop you finding a comfortable position.

This pain can wear you down beyond the hip itself. People with this condition are less likely to be in full-time work than people without it, and they report lower quality of life and more day-to-day disability.

One thing worth knowing: pain on the outside of the hip is not always this condition. If pressing on that bony point does not make you wince, the cause may lie inside the joint instead. That is why an accurate diagnosis matters. Your surgeon will examine you, and may use scans or a numbing injection guided by ultrasound to work out exactly what is causing your pain. Getting the diagnosis right is the key to treating it well.

What's actually happening¶

The bony point on the side of your hip is called the greater trochanter. It works like a pulley. Strong tendons from your buttock muscles run over it, and small fluid-filled cushions sit between them so everything can glide smoothly as you move.

In this condition, those parts become irritated or damaged. The cushions can become inflamed, and the tendons can develop small tears or wear down over time. More than one of these problems is often present at once. Repeated rubbing between the tendon and a tight band of tissue running down the outside of your thigh, along with overuse, injury, or a change in the way you walk, is thought to set this off.

Scans have changed how this condition is understood. Many people told they have bursitis (inflammation of one of those cushions) actually turn out to have a torn or worn tendon instead, with little sign of true bursitis. That matters, because the tendon is the structure doing the real work. It holds your pelvis steady every time you stand, walk, or step, and when it is damaged, the outside of your hip lets you know.

The symptoms you read about above follow directly from this. Tenderness over the bony point comes from the irritated tissue sitting there. Weakness moving your leg out to the side comes from the tendon not pulling properly. Night pain happens because lying on that side presses the sore area directly.

One more thing worth knowing: these problems sit outside the hip joint itself, not inside it. That is why the pain feels different from arthritis, and why getting the diagnosis right matters before any treatment is chosen.

What we can do about it¶

The first step is changing how you load your hip. That can mean easing back on the activities that set your pain off, and adjusting how you walk, stand, or train. Physiotherapy aims to strengthen the buttock muscles that steady your pelvis, through a structured loading program for the gluteal tendons. People who respond to this kind of targeted exercise report less hip pain and better day-to-day function than those who do not follow it. Give this a fair go before moving on. For partial tendon wear, sticking with non-operative care can work well over the long term, with a low risk of the tear getting worse.

If exercise alone is not settling things, we move to medical options. Anti-inflammatory tablets can settle the irritation around the tender spot. Cortisone injections (a steroid medicine placed near the sore tissue) can also calm the pain. More than 60% of surgeons use cortisone as either a first or second step for this problem after a hip replacement, and it works well for bursitis that flares after surgery. It may be less likely to succeed in younger people and those whose legs are different lengths. Injections of platelet-rich plasma (a preparation made from your own blood) have been tested for this condition, with no difference from a placebo up to 6 months afterwards, so we do not use them as a first choice.

Surgery comes into the conversation when non-operative care has not given you enough relief. More than one-third of people do not improve even with the best non-operative treatment, and in those cases operating early can be considered. The operation is done through small cuts using a camera (keyhole surgery). It can release the tight band of tissue running down the outside of your thigh, remove the inflamed cushion, and repair a torn gluteal tendon if one is present. Both keyhole and open tendon repairs lead to functional improvement, with similar failure rates. We will talk through whether surgery suits you, and decide together based on your pain, your scans, and what matters to you.

What to expect¶

For many people, this condition does not simply vanish on its own. The pain tends to persist or keep flaring, especially at night and after activity, unless the underlying cause is treated. Living with it long term takes a real toll: people with this condition are less likely to be in full-time work than people without it, and they report lower quality of life and more day-to-day disability.

The good news is that most people improve when the problem is managed properly. Non-operative care works well for many, particularly for partial tendon wear, where the risk of the tear getting worse is low and long-term results are similar to those reported after surgery. Shock wave therapy (a treatment that delivers sound waves to the sore tissue) can also help. When injections are used, ones placed into the fluid-filled cushion near the tender spot may give longer lasting benefit than others.

Surgery is reserved for people whose symptoms are severe or who have not improved with non-operative care. It relieves symptoms in 95% of severe cases reviewed, and repairs of torn gluteal tendons have held up well over at least 10 years. Improvements in pain and function after keyhole surgery to remove the inflamed cushion are usually noticeable by 1 to 3 months and last throughout follow-up. Where the buttock tendon can no longer do its job at all, a transfer of a nearby tendon has shown reliable improvement in hip function and pain at 3 years.

Recovery is gradual rather than instant. Expect steady gains over weeks to months, not an overnight fix. Your surgeon will talk you through what to watch for. If your hip pain increases or stops improving after surgery, further checks are warranted between 6 and 12 months, and a further operation may be considered at a minimum of 12 months depending on the cause.

One honest caveat: results depend on you as much as on the treatment. Following your postoperative limits matters. One person who reported poor satisfaction after a tendon repair had not stuck to their weight-bearing restrictions, kept smoking against medical advice, and had unrelated spine surgery soon after. Doing your part gives the treatment its best chance.

When to see someone¶

See your GP if you have had pain over the bony point on the side of your hip for more than a few weeks, especially if it is not settling with rest or a change in activity. Ask for a specialist review if pressing on that spot is tender, if your hip feels weak when you move your leg out to the side, or if the pain is waking you at night or making work and daily tasks hard. Getting the right diagnosis early matters, because more than one problem can be present at once and each is treated differently. If you have already had hip surgery, ask for review if the pain increases or stops improving between 6 and 12 months afterwards.

Evidence & references

This is the clinical evidence summary written for health professionals. It is technical, and it lists the research this page was built from. You do not need to read it to understand your treatment or to make a decision about it.

Anatomy & Pathophysiology¶

Bony Anatomy¶

The hip is a multiaxial joint formed by the articulation between the pelvis and femur, connecting the axial skeleton and the lower extremity [8].
The hemipelvis comprises three bones: the ilium, ischium, and pubis, which unite at the triradiate cartilage within the concave acetabulum [8].
The acetabulum comprises an articular crescent-moon-shaped lunate surface and a nonarticular central fossa that serves as the attachment point for the ligamentum teres [8].
The acetabulum is incomplete inferiorly, forming a notch through which vital blood vessels and nerves pass to supply the joint [8].
The femoral head forms two-thirds of a sphere, with a small depression at its center from which the ligamentum teres extends to connect to the acetabular notch [8].
The neck-shaft angle of the femur averages 125° [8].
Normal version, defined as the head-neck angle in the frontal plane, averages 15 to 20° [8].
At the junction of the neck and shaft are the greater and lesser trochanters, which are connected by the intertrochanteric line anteriorly and the intertrochanteric crest posteriorly [8].
The acetabulum is normally anteverted 15 degrees and obliquely oriented in the coronal plane 45 degrees caudally [14].
The posterosuperior articular surface of the acetabulum is thickened to accommodate weight bearing [14].
The inferior surface of the acetabulum contains the acetabular (cotyloid) notch, which is bound by the transverse acetabular ligament [14].
The femoral neck is normally anteverted approximately 14 degrees in relation to femoral condyles, with a range of 1–40 degrees [14].
The femoral neck-shaft angle averages 127 degrees, beginning at 141 degrees in the fetus [14].
The mean femoral neck-shaft angle in the adult is 130° ± 7° [18].
The mean anteversion of the femoral neck is 10° ± 7° [18].
The two prime trabecular groups of the proximal femur are the principal tensile group and the principal compressive group [18].
Secondary compressive and tensile trabecular groups also exist in the proximal femur [18].
The weakest area in the femoral neck is located in the Ward triangle [18].
The calcar femorale is a medial area of dense trabecular bone that transfers stress from the femoral shaft to the inferior portion of the femoral neck [18].

Ligaments and Capsule¶

The hip is surrounded by a dense fibrous capsule extending from the periphery of the acetabulum to the intertrochanteric line of the femoral neck [8].
The capsule enhances joint stability by preventing translation of the femoral head in the acetabulum [8].
The Y-shaped iliofemoral ligament is the thickest and strongest of the three main ligaments supporting the hip [8].
The medial portion of the iliofemoral ligament connects the anterior inferior iliac spine to the anterior intertrochanteric line [8].
The lateral portion of the iliofemoral ligament originates slightly superior to the medial arm and attaches to the anterior greater trochanter [8].
The iliofemoral ligament functions to limit external rotation [8].
In isolation, the lateral arm of the iliofemoral ligament limits extension of the joint [8].
The ischiofemoral ligament extends from the ischial margin of the acetabulum to the greater trochanter of the femur [8].
The ischiofemoral ligament provides support posteriorly and restricts internal rotation motion [8].
The pubofemoral ligament extends from the obturator crest of the pubic bone to the femoral neck [8].
The pubofemoral ligament acts to limit abduction of the joint [8].
Deep fibers from the iliofemoral, ischiofemoral, and pubofemoral ligaments merge to form the zona orbicularis, which circumvents the femoral neck [8].
The hip capsule attaches anteriorly and posteriorly along the periphery of the acetabulum outside the labrum [9].
Inferiorly, the hip capsule is attached to the acetabular labrum [9].
The capsule is attached to the femur anteriorly along the intertrochanteric crest [9].
On the posterior side, the capsule attaches only partially, such that the basicervical region of the femoral neck and the intertrochanteric region of the femur are not intracapsular [9].
The iliofemoral ligament becomes taut in full extension, preventing anterior dislocation and hyperextension of the hip [9].
The twisted orientation of the hip ligaments provides a screw mechanism for the hip in full extension [9].
The ligamentum teres originates in the cotyloid fossa and attaches on the fovea of the femoral head [9].

Labrum¶

The acetabular labrum is a fibrocartilaginous ring attached to the rim of the acetabulum that extends the articulating surface area and increases femoral head coverage [8].
The labrum is triangular in cross section [8].
Only the external one-third of the labrum contains blood vessels, leaving the majority of the structure avascular [8].
The avascular nature of the majority of the labrum limits its healing ability following injury [8].
The labrum is highly innervated, with the presence of both mechanoreceptors and nociceptors [8].
The labrum is absent in the area of the inferior acetabular notch, where the transverse acetabular ligament serves as the continuation of the labrum [8].
The fibrocartilaginous labrum deepens the acetabulum, enhancing stability [5].
Labral functions include load transmission, maintenance of vacuum seal, regulation of synovial fluid hydrodynamics, and joint lubrication [5].

Muscular Anatomy¶

The abductors of the hip are predominantly the gluteus medius and minimus muscles [16].
The gluteus medius has three different components: anterior, middle, and posterior [16].
The gluteus medius and minimus muscles function together to maintain and abduct the femur during the stance phase of gait [16].
A Trendelenburg lurch is an attempt by the body to compensate for abductor weakness by bringing the center of gravity closer to the hip center, forcing the patient to lean toward the affected side [16].
The piriformis muscle originates from the greater sciatic foramen and inserts onto the greater trochanter [16].
The superior gluteal nerve and artery exit the pelvis above the piriformis muscle [16].
The pudendal nerve, internal pudendal artery, nerve to the obturator internus, posterior femoral cutaneous nerve, sciatic nerve, inferior gluteal nerve, inferior gluteal artery, and nerve to the quadratus femoris all exit the pelvis below the piriformis [16].
In 10% of cases, the common peroneal component of the sciatic nerve can pass through the division in the piriformis [16].
The tensor fasciae latae muscle originates laterally on the anterolateral edge of the iliac crest [16].
The action of the tensor fasciae latae is to flex, abduct, and rotate the hip [16].

Neurovascular Anatomy¶

The medial femoral circumflex artery is the main blood supply to the femoral head [18].
The lateral femoral circumflex artery gives rise to the anterior aspect of the extracapsular arterial ring [18].
The superior and inferior gluteal arteries also contribute branches to the extracapsular arterial ring [18].
The ascending cervical arteries originate from the extracapsular arterial ring and are divided into four distinct groups: lateral, medial, posterior, and anterior [18].
The lateral group of ascending branches is the main blood supply to the femoral head [18].
The lateral epiphyseal artery penetrates the femoral head and is believed to be the dominant blood supply to the femoral head from this system [18].
Fractures that disrupt the ascending blood flow to the lateral epiphyseal vessel have an increased risk of osteonecrosis [18].
The artery of the ligamentum teres arises from either the obturator or medial femoral circumflex artery [18].
The artery of the ligamentum teres does not provide sufficient blood supply to maintain the viability of the femoral head [18].
In adulthood, the major blood supply to the femoral head is from the medial femoral circumflex and lateral epiphyseal arteries [21].
The common femoral artery passes anterior and medial to the hip capsule [21].
The common femoral vessels are the most commonly reported extrapelvic vascular structures that are injured during total hip arthroplasty [21].
The most common mechanism of injury to the common femoral vessels is errant retractor placement anterior to the acetabulum [21].
The superior gluteal vessels are branches of the posterior division of the internal iliac artery [21].
Superior gluteal artery injury can occur with the placement of screws in the region of the sciatic notch [21].
The inferior gluteal vessels and internal vessels are branches of the anterior division of the internal iliac artery [21].
The inferior gluteal vessels exit the pelvis between the piriformis and coccygeus muscles [21].

Pathophysiology of Greater Trochanteric Pain Syndrome¶

Greater trochanteric pain syndrome (GTPS) is a conglomerate of conditions including snapping hip, trochanteric bursitis, and gluteal tendinopathy [20].
Lateral-sided hip pain associated with GTPS has been reported to be as debilitating as end-stage degenerative joint disease [20].
In patients between 50 and 79 years of age, GTPS was found in 15% of women and 6.6% of men in one hip [20].
Lateral hip anatomy includes three to four bursae surrounding the side of the hips [20].
The largest bursa is found between the gluteus maximus muscle and the gluteus medius tendon, which is located directly lateral to the greater trochanter [20].
The muscular sheaths and tendinous attachments of the gluteus maximus, iliotibial band, tensor fascia lata, gluteus medius, and gluteus minimus contribute to a complex local environment susceptible to overuse injuries, direct trauma, and gait alterations [20].
Trochanteric bursitis is often related to the other conditions of GTPS and not true inflammation of the local bursa [20].
GTPS encompasses various etiologies of pain at the greater trochanter of the femur, including trochanteric bursitis, gluteus medius or gluteus minimus tendon tears, and friction of the iliotibial band over the greater trochanter [32].
External snapping hip is the result of the iliotibial band snapping over the greater trochanter of the femur [32].

Investigations¶

Clinical Examination and History¶

A thorough understanding of normal anatomy and biomechanics is necessary to identify pathology and determine the appropriate course of treatment for hip pain [1].
A comprehensive clinical examination is required to determine a differential diagnosis because many hip conditions present with similar symptoms [1].
A thorough history is essential to differentiating between common causes of hip pain [1].
Clinical examination tests and imaging findings should be used to confirm a suspected clinical diagnosis [1].

Radiography¶

Conventional radiographs remain critical in the initial imaging evaluation of the hip [2].
Conventional radiographs can be used to diagnose fractures, developmental dysplasia of the hip (DDH), femoroacetabular impingement (FAI), and osteoarthritis [2].
A complete hip series usually consists of an anterior-posterior (AP) pelvis, a centered AP hip, a lateral view (frog-leg, cross-table, Dunn 45° or 90°), and a false-profile (Lequesne) view [2].
Standard AP radiographs of the hip and pelvis are obtained to examine bony architecture, check for evidence of joint space narrowing or changes to bone quality, and quantify femoral head coverage [25].
The Dunn view and frog leg view are appropriate to measure the alpha angle to determine the presence of impingement [25].
Acetabular morphology is assessed on AP pelvis radiographs for acetabular overcoverage and undercoverage [2].
The femoral head-neck junction morphology is often assessed using the alpha angle [2].
Some studies have shown that radiographs, in particular the Dunn 45° view, may be more accurate for determining the alpha angle measurement than CT or MRI [2].
Osteoarthritis of the hip can be categorized using the Kellgren-Lawrence or Tönnis classifications [2].
The Kellgren-Lawrence classification is a 4-point grading system classified into doubtful, mild, moderate, and severe [2].
The Tönnis classification is a 3-point grading system categorized into mild, moderate, and severe [2].
Radiographic and clinical severity do not necessarily correlate, particularly if the radiographs are non-weight-bearing or if false-profile views are not included [2].
The femoral head extrusion index is defined by the length of the femoral head that lies beyond the acetabulum as a percentage of the total horizontal width of the femoral head [2].
Femoral head extrusion index values greater than 25% are considered abnormal [2].
The Tönnis angle is defined by the angle of the acetabular sourcil and a line parallel to the transverse pelvis axis [2].
Tönnis angles between 0° and 10° are considered normal [2].
The lateral center-edge angle, or center-edge angle of Wiberg, is the angle between a line from the center of the femoral head perpendicular to the transverse pelvis axis and a second line from the center of the femoral head to the superolateral most point of the acetabulum [2].
Center-edge angles of 20°-40° are considered normal, while angles from 20° to 25° are considered borderline [2].
An optimal AP pelvis image is required for assessing the "crossover" sign, as pelvic tilt or rotation may lead to false-positive and false-negative results [2].
For neutral pelvic tilt on an AP pelvis radiograph, the sacrococcygeal joint should be between 3 and 5 cm above the superior border of the symphysis pubis [2].
Radiographs remain integral to the assessment of fractures and can be supplemented with CT to further investigate suspected occult fractures, define fracture morphology, and assist in preoperative planning [2].
Radiographs can serially assess hardware positioning and evaluate symptomatic hardware related to open reduction and internal fixation and total hip arthroplasty [2].

Magnetic Resonance Imaging (MRI)¶

MRI is the modality of choice for patients suspected of soft tissue or intra-articular pathology, given its superior sensitivity and specificity [25].
Conventional MRI is effective at identifying osteochondral injuries, musculotendinous pathologies, and inflammation [25].
Magnetic resonance arthrography (MRA) is more appropriate to determine injuries to the labrochondral structures and the ligamentum teres and identify the presence of loose bodies and synovial chondromatosis [25].
The utility of MRA in the accurate detection and staging of articular cartilage lesions is reduced, with sensitivity reported to be less than 50% compared with arthroscopic findings [25].
Recent advances in MRI imaging techniques, such as delayed gadolinium-enhanced MR imaging and T2* mapping, allow for a more in-depth analysis of the structure of articular cartilage [25].
Delayed gadolinium-enhanced MR imaging and T2* mapping were effective at detecting early changes to the articular cartilage surfaces of patients with hip dysplasia and femoroacetabular impingement [25].
MRI provides information regarding the integrity of the acetabular labrum and articular cartilage [3].
The anatomy of the proximal femur as well as the version of the acetabulum and femur may be assessed using MRI [3].
The sensitivity of MRI to acetabular rim chondral lesions is limited [3].
Noncontrast MRI at 3T is generally adequate for diagnosing intra-articular pathology [28].
If 3T imaging is unavailable, MRA can be considered at 1.5T for increased diagnostic accuracy [28].
MRI is helpful in identifying femoral neck stress fracture in athletes and predicting patients that may require surgical intervention [28].
MRI is helpful in assessing complications of conventional and resurfacing hip arthroplasties, particularly those with metal-on-metal bearing systems [28].
Major MRI findings that help predict histologic ALVAL scores include synovial thickening, synovitis, synovial volume, abductor disruption, and soft-tissue edema [28].
A prospective study found similar accuracies between noncontrast 3T MRI and 1.5T MRA in femoroacetabular impingement [26].
A retrospective study evaluating the accuracy of noncontrast 3T MRI versus hip arthroscopy found accuracy for labral tears and acetabular cartilage lesions was 98% and 90%, respectively [26].

Computed Tomography (CT)¶

CT scans are effective for examining cortical and cancellous bone and can be used to create three-dimensional reconstructions of the hip for use in surgical planning [25].
Measurements of femoral head coverage and acetabular and femoral impingement can also be performed reliably using CT images [25].
Low-dose CT with three-dimensional reformats is particularly useful in surgical planning of complex or borderline deformities [3].
CT overcomes the limitations of radiography by providing three-dimensional assessment of bony morphology and, to some degree, assessment of soft-tissue abnormalities [10].
Combined with arthrography, CT can evaluate chondrolabral abnormalities, specifically in patients with contraindications to MRI [10].
CT is helpful in fracture evaluation, particularly in the setting of negative radiographs, or for further defining fracture morphology in patients requiring surgical reduction [10].
The multiplanar and 3D capabilities of CT make it an invaluable tool for assessing bone morphology, but at higher cost and radiation dose [28].
3D volume renderings are useful to aid in preoperative planning in FAI and subspine impingement [28].

Ultrasonography¶

Ultrasonography provides real-time dynamic assessment of the hip and is useful in diagnosing soft-tissue abnormalities about the hip joint, and to a lesser degree, within the hip joint itself [10].
Ultrasonography is particularly useful in providing real-time guidance during diagnostic and therapeutic procedures [10].
Ultrasonography can be an effective modality to identify musculotendinous disruptions, effusions associated with intra-articular pathology, or inflammatory conditions, such as bursitis [25].
Ultrasonography is being increasingly used for targeted injections into muscles, tendons, or intra-articularly around the hip, for use with corticosteroids or biologic treatments [25].
Although ultrasonography is a valuable tool to examine pediatric hip conditions, its utility in evaluating the adult hip is limited [25].
Ultrasonography allows bedside evaluation of the hip and can be used to guide interventions in the office setting [28].
Ultrasonography uses high-frequency sound waves to produce images [29].
A higher frequency ultrasonographic beam can provide higher resolution images of superficial structures such as tendons and ligaments [29].
Doppler ultrasonography can be used to image blood vessels for flow velocity and direction [29].
Elastography is an ultrasonography technique that can assess the elasticity of soft tissues and identify pathology that changes the elasticity of soft-tissue structures, like tendinopathy [29].
Elastography technology is used mostly in research studies but not for clinical use at this point [29].
Ultrasonography is noninvasive at the frequencies used for diagnostic imaging [29].
Ultrasonography equipment is portable and inexpensive compared with MRI and CT equipment [29].
Highly echogenic structures, such as a foreign body that may not be visible on radiographs, can be easily detected using ultrasonography [29].
Ultrasonography can be used to guide targeted therapy, such as injections and ablations, and is useful to guide injections and aspirations [29].
Ultrasonography provides dynamic assessment of structures, such as tendon and nerve subluxation [29].
Image quality and interpretation depend on the experience of the ultrasonography technician and the radiologist [29].
Ultrasonography cannot image inside bone because bone cortex reflects almost all sound waves [29].
Internal joint structures are not well visualized unless they are in a superficial location [29].

References¶

[1] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Summary.

[2] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Radiography.

[3] Aaos Comprehensive Orthopaedic Review 3. Nonarthroplasty Surgical Treatment of the Hip > I. Femoroacetabular Impingement.

[5] Miller S Review Of Orthopaedics. Genetics of musculoskeletal conditions and abnormalities are summarized in Table 1.27 > 2. Arthrology > Hip (Fig. 2.49).

[8] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Osseous and Ligamentous Anatomy.

[9] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > IV. Hip Capsule and Ligaments.

[10] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Introduction.

[14] Miller S Review Of Orthopaedics. SECTION 16 PATELLAR TRACKING IN TOTAL KNEE ARTHROPLASTY > LOWER EXTREMITY.

[16] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > V. Hip Joint Muscles.

[18] Aaos Comprehensive Orthopaedic Review 3. Fractures of the Hip > I. General Considerations.

[20] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Muscular, Neurovascular, and Soft-Tissue Conditions of the Hip > Muscular Conditions About the Hip > Hip Bursitis.

[21] Aaos Comprehensive Orthopaedic Review 3. Surgical Anatomy of the Hip > VI. Neurovascular Structures Surrounding the Hip.

[25] Orthopaedic Knowledge Update 13 Ebook Without Multimedia. Anatomy and Biomechanics, Evaluation, Clinical Examination, and Imaging of the Hip > Imaging.

[26] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Annotated References.

[28] Orthopaedic Knowledge Update Sports Medicine 6. Imaging of the Hip > Summary.

[29] Aaos Comprehensive Orthopaedic Review 3. Musculoskeletal Imaging* > IV. Ultrasonography.

[32] Orthopaedic Knowledge Update Sports Medicine 6. Extra-articular Hip Disorders > Summary.

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